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12-09-2012, 10:53 AM
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Join Date: Dec 2009
Location: Eastern, Alberta
Posts: 887
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Quote:
Originally Posted by Donkey Oatey
Uggg, talk about stubborn. Those 2900 samples where taken in how big of an area? How many deer and elk live in that area?
Now ask the same questions about the areas around the two positive farms. How big of an area? How many animals in that area?
That is how one person figures what is statistically representative.
What hidden agenda do I have? I post the know info from reputable sites. Haven't taken sides. Guess because I don't have some hate for all things government I must have a hidden agenda. What a waste of time.
All I am doing is trying to educate as to the facts that are known. As seen since I jumped in to this train wreck. So many people have no idea about this infection. It doesn't act like anything most people would know. Just trying to clear up misinformation.
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Check your inbox I'm done debating with you, round and round we go
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12-09-2012, 11:35 AM
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Join Date: Apr 2010
Location: Communist Capital of Alberta
Posts: 4,022
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Quote:
Originally Posted by sjemac
I know for a fact that they won't. Repeatedly. So much that I report nothing anymore.
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The officers didn't respond or they didn't lay charges?
I hardly see how doing nothing from now on helps anyone.
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12-09-2012, 12:25 PM
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Join Date: Dec 2009
Location: Alberta
Posts: 3,648
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Quote:
Originally Posted by IHUNT
Baiting is for LAZY hunters!!!!!
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Have to disagree with your train of thought here.. Visit a Sask forum and educate yourself as what is involved in artificial baiting to become successful in harvesting your target prey
Probably as narrow minded as thinking buying a trail cam and utilizing it's features will produce the results you see on the idiot box..
Sheep brought up Cancer, well deeply seeded in our own group this malicious disease is spreading faster than any other concern in our future of this precious resource... We as sportsmen and women are the Cancer and will be the ultimate end to a passion and a future...
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12-09-2012, 12:28 PM
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Join Date: Dec 2009
Location: Eastern, Alberta
Posts: 887
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Quote:
Originally Posted by Donkey Oatey
Guess you don't know much about scientific method. I said the sample size looks small, and it is if you take in to consideration that there are about 50,000 elk and a half million deer in the entire province. But if you look at the local population in the area sampled around the positive farms, it is a fairly representative sample for a localized area. And that is what F&W wanted, a representative sample of the local area around the infected farms to see if the prion had spread to the wild or came from the wild. It didn't.
Sorry can't reasonably debate or have a discussion with someone that sees conspiracies everywhere. And all data can be skewed. Wearing a tinfoil hat won't help you.
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Donkey
One more for sake of arguement your arguement holds no ground , a small high risk area probably similar to the sampling around a cwd pos elk farm as you are suggesting was probably a small high risk area, 486 and 320 tested seems reasonable to believe it was a similar circumstance, what have we learned from these neg tests ? You need numbers when your dealing with low percentages, thousands not hundreds
n late March and early April of 2005 a similar cull took place when a total of 486 deer were culled from what was termed a ?small high-risk area? east of Chauvin. Despite culling these animals, no positive cases of CWD were found.
http://www.meridianbooster.com/2006/...uvin-deer-cull
From this cull in an known small high risk area of 486 animals tested negative.
So if we would have based our results on this low deer submission it's obvious the results are skewed.... agreed?
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12-09-2012, 12:37 PM
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Join Date: Mar 2008
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Quote:
Originally Posted by CBintheNorth
The officers didn't respond or they didn't lay charges?
I hardly see how doing nothing from now on helps anyone.
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They are extremely reluctant to charge a landowner with anything.
Doing nothing certainly avoids me wasting my time and ****ing off my neighbors.
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Moral indignation is a technique used to endow the idiot with dignity.
Marshall McLuhan
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12-10-2012, 07:00 AM
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Join Date: Jan 2009
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[QUOTE=pre64;1740957][QUOTE=buck1979;1740163] (No, Not exactly... I have said that, until I am in a position, that I know where that deer is regularly in daylight, that I will not waste time hunting it, rather continue scouting. Becuase frankly if I dont know where he is spending his daylight hours, I haven't done my homework. The point to that statment is exactly the opposite of how you interperted it. I don't wait, ever.
So if I remove "feeding area" from my comment then I have the correct interpretation. Hardly the exact opposite...Let me put it this way...You locate a shooter buck (trail cam or visually with/without feed/bait) and monitor with trail cams in multiple spots or visually until you locate where he IS travelling in good light, Then you setup and wait - hunt for him over bait or not over bait. The only way I can see you not waiting is if you can walk/stalk right up to the buck and kill him.
[QUOTE=buck1979;1740163] If I hang a trail camera on a post, trail, field edge, or a bait, if I do not get pics of visiuals of my target buck in 48-72 hours, Im in the wrong spot and obviously not in his house. So we scout and move, and figure out the deer, THEN when were in a position where we know where he IS travelling in good light, we will go and hunt him.
Like I said you wait for him to appear - hunt for him. It was an episode of Canada in the Rough (stickers buck) where you made those comments, however you never mentioned "his house" so I took it you were referencing bait. "stickers" was killed over bait. I knew of a 230 inch non-typical, problem was his bedroom was in the middle of a 2 sq. mile solid thick bush "house". So I guess the term "house" is relative to the area the bucks lives in and how to hunt him. He was eventually killed (no bait involved) out on the edge of his "house" after 2 visual sightings (homework) and 3 hunting seasons of waiting/hunting the edge he favored.
[QUOTE=buck1979;1740163] ...I read on this thread with the right bait you can grow a 200" whitetail... I will go with umm... no.
I did not say "with the right bait you can grow a 200" whitetail".
I said "with a few years of feeding prime, genetically superior bucks the right "$tuff", you could grow a 200+ inch Sask WT..."could" being the operative word, as in possibly, not "can" as you quoted me...big difference there.
[QUOTE=buck1979;1740163] You cant grow a wild whitetail period.
Depends on what defines a truly wild whitetail. Once you start placing (not growing/deriving from the ground) feed/bait/minerals etc. repeatedly for whitetails at a specific small spot, the deer that feed there are not 100% wild. Even though they may be legally killed in some provinces/states. Less than 100% wild, the potential thru supplemental artificially placing feed to influence and enhance antler size of certain bucks is a fact. Optimum conditions for antler growth, whether they occur naturally or are created/placed artificially, can produce bigger antlers. For 15 years in the 1980's and 1990's I made available supplemental feed to the local (no hunting zone) deer around my place. Usually had around 75 deer annually. I watched many bucks mature and grow up from age 1 1/2 including a couple of 180 class and a 190 class typical and a 240 class non-typical. At the start I fed them only from Dec thru April. When I started supplemental feeding during May thru July for a few years 2 bucks really put on the inches and grew their best racks, becoming 240 class non-typical and 180 class typical bucks even though we had dry spring-dry summer conditions. When they were young there antlers were nothing special, unlike an 8 1/2 yr. old 190 class typical who was approx. a 100 inch 10 pt buck when he was 1 1/2. Many other bucks also had better than ave. yearly increases in antler growth when I fed them during the growing season. But many bucks never passed the 150-160 class.
Pre64, I'm not argueing with you, but I think your just reading what you want to hear. Your first post was specificaly meant to sound like dump a bait and wait. I was pointing out, thats exactly what we try to show guys not to do. If there is a buck you want to kill, I dont care if its in a 25x25 block of timber, if you dont have a knowledge of where he is in daylight evey other day, you just dont have enough info about the deer. period. I dont believe in nocturnal deer. This....is just my opinion. There are several bucks its taken us years to get. In the end it was always our errors or lack of knowledge that prevented us from getting him. I hunted a buck in 2011, I hunted him 70 nights, with one poor encounter. This year we put more scouting in to the point that I slept in the fields in Aug to be there to try and spot him coming into bedding etc... We changed everything and re-set up. I had 3 encounters with him and killed him on the 4th night. I shot him Oct 6th, so 37 days into the season. In 37days there were only 4 that the conditions were right as we were set up within 200yds of where he was bedding. My point is, bait and wait is very un productive.
In Sk, everywhere is a food plot, you cannot artificially feed enough to change their horn growth. I dont buy it for a second. THey have optimal nutrition on every quater of land. I know many guys that have tried to "grow" them to no avail.
Last edited by buck1979; 12-10-2012 at 07:06 AM.
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12-10-2012, 09:42 AM
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Join Date: Mar 2008
Location: Alberta
Posts: 4,278
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Quote:
Originally Posted by buck1979
In Sk, everywhere is a food plot, you cannot artificially feed enough to change their horn growth. I dont buy it for a second. THey have optimal nutrition on every quater of land. I know many guys that have tried to "grow" them to no avail.
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Not to mention, in sask they have rules about how much bait you use, and the time frame you can use it. You aren't growing a 200" in 3months, and you certainly aren't keeping a specific deer in the area year round.
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12-10-2012, 09:43 AM
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[QUOTE=buck1979;1744568][QUOTE=pre64;1740957][QUOTE=buck1979;1740163] (No, Not exactly... I have said that, until I am in a position, that I know where that deer is regularly in daylight, that I will not waste time hunting it, rather continue scouting. Becuase frankly if I dont know where he is spending his daylight hours, I haven't done my homework. The point to that statment is exactly the opposite of how you interperted it. I don't wait, ever.
So if I remove "feeding area" from my comment then I have the correct interpretation. Hardly the exact opposite...Let me put it this way...You locate a shooter buck (trail cam or visually with/without feed/bait) and monitor with trail cams in multiple spots or visually until you locate where he IS travelling in good light, Then you setup and wait - hunt for him over bait or not over bait. The only way I can see you not waiting is if you can walk/stalk right up to the buck and kill him.
[QUOTE=buck1979;1740163] If I hang a trail camera on a post, trail, field edge, or a bait, if I do not get pics of visiuals of my target buck in 48-72 hours, Im in the wrong spot and obviously not in his house. So we scout and move, and figure out the deer, THEN when were in a position where we know where he IS travelling in good light, we will go and hunt him.
Like I said you wait for him to appear - hunt for him. It was an episode of Canada in the Rough (stickers buck) where you made those comments, however you never mentioned "his house" so I took it you were referencing bait. "stickers" was killed over bait. I knew of a 230 inch non-typical, problem was his bedroom was in the middle of a 2 sq. mile solid thick bush "house". So I guess the term "house" is relative to the area the bucks lives in and how to hunt him. He was eventually killed (no bait involved) out on the edge of his "house" after 2 visual sightings (homework) and 3 hunting seasons of waiting/hunting the edge he favored.
[QUOTE=buck1979;1740163] ...I read on this thread with the right bait you can grow a 200" whitetail... I will go with umm... no.
I did not say "with the right bait you can grow a 200" whitetail".
I said "with a few years of feeding prime, genetically superior bucks the right "$tuff", you could grow a 200+ inch Sask WT..."could" being the operative word, as in possibly, not "can" as you quoted me...big difference there.
Quote:
Originally Posted by buck1979
You cant grow a wild whitetail period.
Depends on what defines a truly wild whitetail. Once you start placing (not growing/deriving from the ground) feed/bait/minerals etc. repeatedly for whitetails at a specific small spot, the deer that feed there are not 100% wild. Even though they may be legally killed in some provinces/states. Less than 100% wild, the potential thru supplemental artificially placing feed to influence and enhance antler size of certain bucks is a fact. Optimum conditions for antler growth, whether they occur naturally or are created/placed artificially, can produce bigger antlers. For 15 years in the 1980's and 1990's I made available supplemental feed to the local (no hunting zone) deer around my place. Usually had around 75 deer annually. I watched many bucks mature and grow up from age 1 1/2 including a couple of 180 class and a 190 class typical and a 240 class non-typical. At the start I fed them only from Dec thru April. When I started supplemental feeding during May thru July for a few years 2 bucks really put on the inches and grew their best racks, becoming 240 class non-typical and 180 class typical bucks even though we had dry spring-dry summer conditions. When they were young there antlers were nothing special, unlike an 8 1/2 yr. old 190 class typical who was approx. a 100 inch 10 pt buck when he was 1 1/2. Many other bucks also had better than ave. yearly increases in antler growth when I fed them during the growing season. But many bucks never passed the 150-160 class.
Pre64, I'm not argueing with you, but I think your just reading what you want to hear. Your first post was specificaly meant to sound like dump a bait and wait. I was pointing out, thats exactly what we try to show guys not to do. If there is a buck you want to kill, I dont care if its in a 25x25 block of timber, if you dont have a knowledge of where he is in daylight evey other day, you just dont have enough info about the deer. period. I dont believe in nocturnal deer. This....is just my opinion. There are several bucks its taken us years to get. In the end it was always our errors or lack of knowledge that prevented us from getting him. I hunted a buck in 2011, I hunted him 70 nights, with one poor encounter. This year we put more scouting in to the point that I slept in the fields in Aug to be there to try and spot him coming into bedding etc... We changed everything and re-set up. I had 3 encounters with him and killed him on the 4th night. I shot him Oct 6th, so 37 days into the season. In 37days there were only 4 that the conditions were right as we were set up within 200yds of where he was bedding. My point is, bait and wait is very un productive.
In Sk, everywhere is a food plot, you cannot artificially feed enough to change their horn growth. I dont buy it for a second. THey have optimal nutrition on every quater of land. I know many guys that have tried to "grow" them to no avail.
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Sk, everywhere is a food plot, you cannot artificially feed enough to change their horn growth. I dont buy it for a second. THey have optimal nutrition on every quater of land. I know many guys that have tried to "grow" them to no avail.
So then how would you explain the Farmed animals and their horn growth?
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12-10-2012, 10:02 AM
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Join Date: Jun 2009
Posts: 141
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Quote:
Originally Posted by justinO
After researching yesterday your two answers are opinion based no one knows When CWD started, How it started or how exactly it is transferred. To assume it has not been around for milenia is just an opinion same as how it is spread through BAITING like so many have said on this thread, not one scientist has any proof its just an opinion. The only fact is a very small percentage of deer and elk have it and the first time it was found was in the 60's as this was when testing started. If this is the case how in 40+ years has CWD not spread throughout north America like cancer as some would say? What has more of an affect on our Game population CWD or the poor management from our province such as killing all the doe's in Alberta?
But I have to tapp out on researching this issue as I have my own 50 page thesis paper to write, and all I have found to my questions is the same answer it is unknown.
If someone continues researching this topic and can find factual information as to WHERE CWD originated? (Not in 1967 because of a test I know this already) How CWD originated? How CWD is transferred? And if this is a natural disease or something created through human involvement.
Please P.M me
Thanks in Advance
Justin
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Exactly!!!!!!
Same as cruetsfelt Jacobs diease, Same as Variance cuetlsfelt Jacobs disease, same as Bovine Spongeiform Encephelopathy AKA: BSE AKA: Mad Cow Disease.
Is it possible CWD is a naturally occuring disease in wild ungulate populations and only because our current ungulate populations are at a historic all time high we are seeing an increase in reported cases?
...Probably not because SRD doesn't think so and they are a bunch of geniuses...you know, same as Alberta can't support a Grizzley hunt and Bighorn sheep are on the Brink!!!!
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12-10-2012, 10:14 AM
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Join Date: Jun 2010
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Quote:
Originally Posted by saskfly
Exactly!!!!!!
Same as cruetsfelt Jacobs diease, Same as Variance cuetlsfelt Jacobs disease, same as Bovine Spongeiform Encephelopathy AKA: BSE AKA: Mad Cow Disease.
Is it possible CWD is a naturally occuring disease in wild ungulate populations and only because our current ungulate populations are at a historic all time high we are seeing an increase in reported cases?
...Probably not because SRD doesn't think so and they are a bunch of geniuses...you know, same as Alberta can't support a Grizzley hunt and Bighorn sheep are on the Brink!!!!
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Once again missing the point. vCJD is most assuredly caused by consuming BSE infected at risk materials. Yes there are natural prion diseases. In humans there are
* Creutzfeldt-Jakob Disease (CJD)
* Variant Creutzfeldt-Jakob Disease (vCJD)
* Gerstmann-Straussler-Scheinker Syndrome
* Fatal Familial Insomnia
* Kuru
In animals there are:
* Bovine Spongiform Encephalopathy (BSE)
* Chronic Wasting Disease (CWD)
* Scrapie
* Transmissible mink encephalopathy
* Feline spongiform encephalopathy
* Ungulate spongiform encephalopathy
Now just because they are all prion disease does not mean they transmit the same way or can even be transmitted. CJD and vCJD are non-infectious. You can't get them from shaking hands or licking butts or anything.
BSE can not be transmitted other than by consuming contaminated parts ie, brain, spinal column etc.
Kuru is the same way. Eat the brain of a Kuru infected person.
Scrapie on the other hand is infectious. Like CWD. It can spread live animal to live animal.
That is why those two diseases are treated differently than the others.
BSE is easy to eradicate. So is Kuru. Don't be a cannibal.
Bah why do I do this to myself?
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12-10-2012, 10:22 AM
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Join Date: Jun 2009
Posts: 141
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"If someone continues researching this topic and can find factual information as to WHERE CWD originated? (Not in 1967 because of a test I know this already) How CWD originated? How CWD is transferred? And if this is a natural disease or something created through human involvement."
Donkey,
Are the answers to the above questions known and if they are, are they accepted as scientific fact? Is there anywhere I can read the accepted studies?
I am just interested in the facts. If you can point me in the right direction it would be greatly appreciated.
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12-10-2012, 10:28 AM
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Quote:
Originally Posted by Donkey Oatey
Bah why do I do this to myself?
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LOL...you have far more patience than I.....
You are bringing some great info to the discussion....while lost on a few of the posters here, I suspect the silent majority is taking it in Keep up the good work!
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12-10-2012, 10:58 AM
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Join Date: Jun 2010
Posts: 2,264
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Quote:
Originally Posted by saskfly
"If someone continues researching this topic and can find factual information as to WHERE CWD originated? (Not in 1967 because of a test I know this already) How CWD originated? How CWD is transferred? And if this is a natural disease or something created through human involvement."
Donkey,
Are the answers to the above questions known and if they are, are they accepted as scientific fact? Is there anywhere I can read the accepted studies?
I am just interested in the facts. If you can point me in the right direction it would be greatly appreciated.
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Classic CJD http://www.cdc.gov/ncidod/dvrd/cjd/index.htm
vCJD http://www.cdc.gov/ncidod/dvrd/vcjd/index.htm (talks about how they know its from BSE)
Kuru http://www.nlm.nih.gov/medlineplus/e...cle/001379.htm
CWD interesting little paper. http://www.google.ca/url?url=http://...0Z8NzC1RES94BA
No one can definitively say where CWD comes from. There are three theories. Scrapie jumped the species barrier in the research facility in Colorado (where the disease was first identified, with mule deer housed in the same pens as scrapie infected sheep once housed), it is a sporadic disease that has appeared and disappeared, or its always been there but no one was looking for it.
If its natural then we should do nothing. Let it runs its course I guess. Bury heads in sand. Do nothing.
That doesn't sound right to me. Does it to anyone else? Research, and maybe just maybe we will find answers. Maybe a vaccine. Maybe the source. Maybe nothing.
What we do know is that CWD is spread from animal to animal without having to ingest infected brain or spinal column like BSE or Kuru. It can be spread live animal to live animal and it can stay in the environment for a really really long time.
So from that we can say that lower the density of animals in any given spot should slow the disease. One thing we have going for us in Alberta verses Wisconsin, Colorado and others. Don't artifically group animals in close areas, (baiting) where the concentration of the prion can increase. Seems to me prevention is worth more than anything.
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12-10-2012, 11:13 AM
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Join Date: Jan 2009
Posts: 39
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Quote:
Originally Posted by pikeslayer22
So then how would you explain the Farmed animals and their horn growth?
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Controlled enviroments. Not only can you control what they eat.... you can control what they dont. Stop a wild deer from browsing, feeding in an alfalfa field. That, steriod and growth hormone injections, and decades of genetic control through intensive breeding.
Even in a farm deer, you cant turn a 150 into a 200. They can only grow what they have in their genetic make up to grow. Hormones and growth medications etc can be administered to make him reach his genetic potential but.. he is what he is. Some giant 300" cactus head from a farm, if let into the wild, would shed and still regrow a giant.
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12-10-2012, 11:26 AM
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Join Date: Dec 2009
Location: Eastern, Alberta
Posts: 887
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Quote:
Originally Posted by ishootbambi
thats some interesting arithmetic. the real numbers that matter are infected animals in number of heads tested. in both alberta and saskatchewan, infection rates are running less than 1%.....actually pretty close to a half of one percent.
actually david, you may need to catch up. colorado has been dealing with this for 45 years now, and they have deer densities around 10 times higher than we do....not to mention elk at around 30 times what we have. in 30 years, we will likely have around a 2% infection if you extropolate the numbers based on what has been observed in the state that has dealt with this the longest. one particualr thing to note is that there is still a small pocket of colorado that has ZERO infection after all this time.
i find it best to examine available data from sources based in colorado and wyoming and not the BS that srd in alberta is peddling. to do nothign is no worse than standing by....given that NOTHING tried so far has had any effect. you could argue that the culls are actually doing damage to the herds. there has never been 100% infection even in captive herds. many experts believe that some animals are immune to this disease, and by culling thousands of healthy deer, you are actually removing those safe animals. still just a theory, but whats proven is that culls are completly inneffective because of the persistence of the disease long after the host is gone. your ebola example works well for viral infections...but this is very different.
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Keep up the good work Dale........
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12-10-2012, 01:24 PM
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Location: CANADA
Posts: 6,287
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SRD did a slide presention to ABA
David
CWD UPDATE --- We were shown a power point outlining some information of CWD, the current strategy, creating a National Strategy (a broad program and establish best practices) and what the future may hold. CWD is 10x more prevalent in mule deer than whitetails, 2x more so in males, is not native to deer/elk/moose, Alberta has had 127 positives out of 45000 samples, CWD is spreading and evidence from other areas suggest it can lead to long term declines in populations and hunting opportunity, feeding and baiting deer can increase spread.
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12-10-2012, 01:55 PM
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Join Date: Dec 2009
Location: Alberta
Posts: 3,648
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Quote:
Originally Posted by Speckle55
SRD did a slide presention to ABA
David
CWD UPDATE --- We were shown a power point outlining some information of CWD, the current strategy, creating a National Strategy (a broad program and establish best practices) and what the future may hold. CWD is 10x more prevalent in mule deer than whitetails, 2x more so in males, is not native to deer/elk/moose, Alberta has had 127 positives out of 45000 samples, CWD is spreading and evidence from other areas suggest it can lead to long term declines in populations and hunting opportunity, feeding and baiting deer can increase spread.
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Thanks David,
Last line makes me smile a little actually, mule deer subject to 10 fold the target species for baiting with a confirmed percentage of infection 0.0028.. So move decimal one more notch to the right and 0.00028 chance of a target species such as the whitetail deer possibly having an increased risk of disease if Alberta allowed baiting for the purpose of hunting.
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12-10-2012, 02:02 PM
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If you did like we do with rats on the border area for 14 to 16 years then the prion infect rate would be greatly affected
sure in the infected zone there will be very few deer but the rate of infection wiil be able to be identified and 100 percent killing of animals in those very hot areas will be have to be done
nothing differn,t than the CDC putting a Quarintine on a area for public safty and allowing no movement till they get a handle to the outbreak
have a great years of hunting all the other non infected WMU's and even if this does not work you will have not done any thing differn,t than what will happen if you do nothing as in the end there will be no deer left untouched in the infected areas /hot spots
if not it will take it's course and soon within 30 more years every WMU in Alberta will have CWD and it will be at the mountains and will have jumped to ELK and MOOSE and will jump too Sheep in afew years from then
is this what you guys want the next paragraph
but no do not whatever you do try to keep in at the border we know that won,t help is that what your saying and keep the numbers high so that the infection rate is quicker and more animals get CWD and so that the travelers
can run the major corridors and go further into more WMU's
if you look at Colarado it is mostly red in my previous post and the numbers in some areas is allmost non-existant as CWD has taken its course now they will have to make 14 to 20 years to come back with none infected deer or it will stay that low
pretty easy for me too say lets be proactive and do something not now but Right Now
i hope we have the balls to do the program this time and inform the public that this is a CDC/WHO type thing in layman terms
I'am all in
Food for Thought
David
David
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12-10-2012, 02:08 PM
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Quote:
Originally Posted by Sledhead71
Thanks David,
Last line makes me smile a little actually, mule deer subject to 10 fold the target species for baiting with a confirmed percentage of infection 0.0028.. So move decimal one more notch to the right and 0.00028 chance of a target species such as the whitetail deer possibly having an increased risk of disease if Alberta allowed baiting for the purpose of hunting.
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Is that new math?
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12-10-2012, 02:10 PM
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Join Date: Dec 2010
Location: CANADA
Posts: 6,287
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Quote:
Originally Posted by Sledhead71
Thanks David,
Last line makes me smile a little actually, mule deer subject to 10 fold the target species for baiting with a confirmed percentage of infection 0.0028.. So move decimal one more notch to the right and 0.00028 chance of a target species such as the whitetail deer possibly having an increased risk of disease if Alberta allowed baiting for the purpose of hunting.
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can i ask you a question? .. what do you think we should do differn,t than the National Program that Trent Bollinger and assocates suggest
David
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12-10-2012, 02:11 PM
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Join Date: Dec 2010
Location: Look behind you :)
Posts: 27,818
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Quote:
Originally Posted by sheephunter
Is that new math?
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It is math.....it is not like it is an exact science
LC
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12-10-2012, 02:14 PM
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Join Date: Dec 2009
Location: Alberta
Posts: 3,648
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Quote:
Originally Posted by sheephunter
Is that new math?
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LOL,
See you can manipulate numbers to show others there is a concern
Glad you caught that, was my intention to skew the masses, a little like the ones who produced this information indicating in last line that baiting is certainly going to increase the risk
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12-10-2012, 02:20 PM
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Quote:
Originally Posted by Sledhead71
LOL,
See you can manipulate numbers to show others there is a concern
Glad you caught that, was my intention to skew the masses, a little like the ones who produced this information indicating in last line that baiting is certainly going to increase the risk
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Baiting is going to increase risk of disease in our whitetails...that is an undisputable fact. I guess we could throw calculations around all day as to the percentage of the increase in risk but the fact that it increases risk is good enough for me to say no.
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12-10-2012, 03:09 PM
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Join Date: Dec 2009
Location: Eastern, Alberta
Posts: 887
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Quote:
Originally Posted by sheephunter
Baiting is going to increase risk of disease in our whitetails...that is an undisputable fact. I guess we could throw calculations around all day as to the percentage of the increase in risk but the fact that it increases risk is good enough for me to say no.
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If this is indeed fact, then why are we still elk and deer farming province wide and especially in proven cwd positive zones? Knowing full well wild deer and wild elk can come in contact with domestic concentrated elk and deer farms?
What is wrong with this picture?........
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12-10-2012, 03:12 PM
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Quote:
Originally Posted by NIKON
If this is indeed fact, then why are we still elk and deer farming province wide and especially in proven cwd positive zones? Knowing full well wild deer and wild elk can come in contact with domestic elk and deer farms?
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That's a very good question indeed.
I assume you are a game farmer and I assume you'd like to see hunt farms in Alberta and I assume you wouldn't like to see the connection between game farms and CWD in wild ungulates made.
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12-10-2012, 03:17 PM
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Join Date: Dec 2009
Location: Eastern, Alberta
Posts: 887
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Quote:
Originally Posted by sheephunter
That's a very good question indeed.
I assume you are a game farmer and I assume you'd like to see hunt farms in Alberta and I assume you wouldn't like to see the connection between game farms and CWD in wild ungulates made.
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Exact opposite, it's a poke at our srd and the whole problem with the gov in our province
Tap root cwd, nuff said....... It's called politics and who's gonna compensate the farmer when they finally wake up and realize we are just chasing our tail
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12-10-2012, 03:33 PM
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Quote:
=NIKON;1745141]Exact opposite
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Actually I figured that...I was just demonstrating how annoying it was when someone assumes a bunch of things about you that are false
Last edited by sheephunter; 12-10-2012 at 03:42 PM.
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12-10-2012, 04:14 PM
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Join Date: Dec 2009
Location: Eastern, Alberta
Posts: 887
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Quote:
Originally Posted by sheephunter
Actually I figured that...I was just demonstrating how annoying it was when someone assumes a bunch of things about you that are false
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LOL
This is what i find annoying so i will ask you this?
Yes or NO, do you feel 100% confident cwd doesn't exist west of the border? I;m talking as far west as Edmonton near or around pos elk and deer farms that have tested positve for cwd.....knowing full well it exists in the soil for many years and knowing the gov only tested a few hundred heads and random road kills...... Also knowing with very low percentages of actual cwd positives we need thousands of head submissions to have accurate results.... I'm not talking province wide but around the pos cwd deer and elk farms in Alberta? Also knowing a deer can travel up as far as 40kms between winter and summer ranges proven by collared deer in the cwd zone... Yes or NO?
I want your own personal opinion , not an opinion swayed by _____
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12-10-2012, 04:20 PM
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Double tap
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12-10-2012, 04:21 PM
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Quote:
Originally Posted by NIKON
LOL
This is what i find annoying so i will ask you this?
Yes or NO, do you feel 100% confident cwd doesn't exist west of the border? I;m talking as far west as Edmonton near or around pos elk and deer farms that have tested positve for cwd.....knowing full well it exists in the soil for many years and knowing the gov only tested a few hundred heads and random road kills...... Also knowing with very low percentages of actual cwd positives we need thousands of head submissions to have accurate results.... I'm not talking province wide but around the pos cwd deer and elk farms in Alberta? Also knowing a deer can travel up as far as 40kms between winter and summer ranges proven by collared deer in the cwd zone... Yes or NO?
I want your own personal opinion , not an opinion swayed by _____
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IMHO....it could be anywhere that we have game farms. No need to add a caveat...my opinion is never swayed by anything but personal research and experience....it'll save you some typing next time
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